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Im Asian and turn red very easily after drinking. I take a Pepcid AC before drinking and I don't turn red at all unless I drink excessively.
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yup. people always thinkin your all f*cked up cuz your red, but i get mad red after 1 beer.Originally Posted by shatterkneesinc
i get red but no where near drunk
people get the impression if you are blushing you are drunk
that alsoOriginally Posted by freakydestroyer
there's a way around it I dunno what it was? maybe it was pepcid AC
Originally Posted by K2theAblaM
It's the pigmentation of our skin color. We get flushed red due to the increased blood flow since alcohol thins the blood.
It's also why Asian babies are bright red when we're born.
Many Orientals lack the mitochondrial aldehyde dehydrogenase (ALDH2) activity responsible for the oxidation of acetaldehyde produced during ethanol metabolism. These individuals suffer the alcohol-flush reaction when they drink alcoholic beverages. The alcohol-flush reaction is the result of excessive acetaldehyde accumulation, and the unpleasant symptoms tend to reduce alcohol consumption. The subunit of this homotetrameric enzyme was sequenced and the abnormality in the inactive enzyme shown to be a substitution of lysine for glutamate at position 487. We have used the polymerase chain reaction to determine the genotypes of 24 livers from Japanese individuals. Correlating genotype with phenotype leads to the conclusion that the allele (ALDH2(2)) encoding the abnormal subunit is dominant.
Originally Posted by Dirtylicious
Originally Posted by K2theAblaM
It's the pigmentation of our skin color. We get flushed red due to the increased blood flow since alcohol thins the blood.
It's also why Asian babies are bright red when we're born.
no
http://www.pubmedcentral....render.fcgi?artid=303676
Many Orientals lack the mitochondrial aldehyde dehydrogenase (ALDH2) activity responsible for the oxidation of acetaldehyde produced during ethanol metabolism. These individuals suffer the alcohol-flush reaction when they drink alcoholic beverages. The alcohol-flush reaction is the result of excessive acetaldehyde accumulation, and the unpleasant symptoms tend to reduce alcohol consumption. The subunit of this homotetrameric enzyme was sequenced and the abnormality in the inactive enzyme shown to be a substitution of lysine for glutamate at position 487. We have used the polymerase chain reaction to determine the genotypes of 24 livers from Japanese individuals. Correlating genotype with phenotype leads to the conclusion that the allele (ALDH2(2)) encoding the abnormal subunit is dominant.